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NaV1.5 sodium channels increase breast cancer cell invadopodial activity by both controlling Src kinase-dependent F-actin polymerization and promoting NHE-1-dependent proton efflux and extracellular matrix digestion

Bibliographic reference Brisson, Lucie ; Driffort, Virginie ; Benoist, Lauriane ; Poet, Mallorie ; Counillon, Laurent ; et. al. NaV1.5 sodium channels increase breast cancer cell invadopodial activity by both controlling Src kinase-dependent F-actin polymerization and promoting NHE-1-dependent proton efflux and extracellular matrix digestion.4th annual meeting of the ISPDC (Munich, Germany, du 10/10/2013 au 12/10/2013). In: Frontiers in Pharmacology,
Permanent URL http://hdl.handle.net/2078/154586