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Type 2 immune response associated with silicosis is not instrumental in the development of the disease.
Primary tabs
Document type | Article de périodique (Journal article) – Journal Article, Research Support, Non-U.S. Gov't |
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Access type | Accès restreint |
Publication date | 2007 |
Journal information | "American journal of physiology. Lung cellular and molecular physiology" - Vol. 292, no. 1, p. L107-13 (2007) |
Peer reviewed | yes |
issn | 1040-0605 |
Publication status | Publié |
Affiliations |
UCL
- MD/MNOP - Département de morphologie normale et pathologique UCL - MD/ESP - Ecole de santé publique UCL - (SLuc) Service de biochimie médicale UCL - (MGD) Service d'anatomie pathologique |
MESH Subject | Animals ; Bronchoalveolar Lavage Fluid - immunology ; Disease Models, Animal ; Female ; Immunoglobulin G - metabolism ; Interleukin-13 - metabolism ; Interleukin-4 - deficiency - genetics ; Lung - immunology - pathology ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Receptors, Cell Surface - deficiency - genetics ; Silicosis - etiology - immunology - pathology ; Th1 Cells - immunology ; Th2 Cells - immunology |
Links |
Bibliographic reference | Misson, Pierre-Damien ; Brombacher, Frank ; Delos, Monique ; Lison, Dominique ; Huaux, François. Type 2 immune response associated with silicosis is not instrumental in the development of the disease.. In: American journal of physiology. Lung cellular and molecular physiology, Vol. 292, no. 1, p. L107-13 (2007) |
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Permanent URL | http://hdl.handle.net/2078.1/10717 |